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Aztron Medtech

Aztron Medtech offers the TendMIN® system, a percutaneous, ultrasound‑guided platform for Achilles tendon repair that uses incisions of 1 cm or less to preserve the paratenon and minimize soft‑tissue trauma.

Newark, DelawareFounded 202151K+ followers
Updated 2 months ago

Funding

Funding not disclosed

Funding rounds are not available yet.

Founders

Founder details are not available yet.

Product

Problem

Achilles tendon ruptures are traditionally treated with open surgery, which requires large incisions, extensive soft‑tissue dissection, and paratenon disruption. This leads to increased postoperative pain, scarring, longer rehabilitation, and higher risk of complications.

Solution

Aztron Medtech’s TendMIN® system delivers a percutaneous, ultrasound‑guided approach for Achilles tendon repair through incisions of 1 cm or less. The platform preserves the paratenon and minimizes soft‑tissue trauma while enabling precise suture placement at optimal tendon locations. Surgeons can select from locking, non‑locking, or anchor‑based configurations, maintaining repair strength comparable to open techniques. The procedure is streamlined to take roughly half the time of conventional surgery, facilitating faster patient recovery and earlier return to function.

Target Audience

Primary users are orthopedic surgeons and sports‑medicine specialists who perform Achilles tendon repairs, particularly in hospitals and ambulatory surgical centers seeking minimally invasive solutions.

Features

  • Ultrasound‑guided visualization for accurate suture placement without large incisions
  • <1 cm percutaneous access that preserves the paratenon and reduces tissue trauma
  • Supports multiple repair strategies: end‑to‑end, locking constructs, and calcaneal anchor fixation
  • Compatible with standard sutures and suture tape, offering flexibility in construct selection
  • Designed for rapid execution, achieving repair times up to 50% faster than open surgery
  • Minimal scarring and lower postoperative pain due to reduced soft‑tissue disruption
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